Identifying Skin Cancer Cures and Medical Manifestations

Journal of Cancer Clinical Trials

ISSN: 2577-0535

Open Access

Perspective - (2022) Volume 7, Issue 5

Identifying Skin Cancer Cures and Medical Manifestations

Marcela Miles*
*Correspondence: Marcela Miles, Centre of Biomedical Studies, Girona, Spain, Email:
Centre of Biomedical Studies, Girona, Spain

Date of Submission: 09-May-2022, Manuscript No. jcct-22-64017; Editor assigned: 09-May-2022, Pre QC No. P-64017; Reviewed: 21-May-2022, QC No. Q-64017; Revised: 25-May-2022, Manuscript No. R-64017; Published: 31-May-2022 , DOI: 10.37421/2577-0535.2022.7.165
Citation: Miles, Marcela. “Identifying Skin Cancer Cures and Medical Manifestations.” J Cancer Clin Trials 7 (2022): 165.
Copyright: © 2022 Miles M. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.


Skin cancer can develop in either basal or squamous cells. The most frequent kinds of skin cancer are basal cell carcinoma and squamous cell carcinoma. Non-melanoma skin cancer is another name for it. Actinic keratosis is a type of keratosis that can progress to squamous cell cancer. Melanoma is uncommon compared to basal cell carcinoma and squamous cell carcinoma. It has a higher chance of infiltrating surrounding tissues and spreading to other sections of the body. When skin cells age and die, or when they get injured, new skin cells form. When this process fails, a fast proliferation of cells (some of which may be aberrant cells) occurs. This group of cells could be noncancerous (benign), meaning they don't spread or harm you, or cancerous, meaning they can spread to neighbouring tissue or other parts of your body if not discovered and treated early [1].


During a medical procedure, the tumour and surrounding tissue are surgically removed. During a simple surgical operation, many skin malignancies can be promptly and simply removed from the skin. In many cases, no additional therapy is required. Skin cancer occurs when the body does not repair damage to the DNA inside skin cells, allowing the cells to divide and grow uncontrollably. Skin cell damage may be caused by a variety of factors, including genetics and skin type. Most cases of skin cancer result from overexposure to Ultraviolet (UV) light, including sunlight and tanning beds, with the risk growing with the amount of exposure [2].

The majority of these operations begin with a local anaesthetic to numb the skin. They can be performed at a dermatologist's office, a surgical oncologist's office, a general surgeon's office, a plastic surgeon's office, a nurse practitioner's office, or a physician assistant's office. Other procedures, including as more comprehensive broad excisions and sentinel lymph node biopsies, are done under local or general anaesthesia in a hospital operating room. Merkel cell cancer is frequently treated in this manner. The majority of skin malignancies are treated with surgery. A dermatologist or other trained clinician may perform an outpatient procedure utilising a local anaesthetic for patients with basal cell or squamous cell carcinomas. The cancer cells are removed, along with a tiny portion of surrounding skin, known as the margin, in these operations, as in most skin cancer surgeries [3-5].


Nonsurgical treatments may be utilised to eliminate or destroy localised skin cancer cells in some circumstances. These procedures can be used to treat early-stage basal cell or squamous cell carcinomas, as well as noncancerous or precancerous lesions, either alone or in combination with other treatments. The following are examples of topical therapies. In order to eliminate cancer cells, this procedure combines photosensitive medicine with light. A light-sensitive chemical, commonly aminolevulinic acid, is applied directly to the tumour in this approach. The treated region is exposed to a specific blue light that activates the drug and targets cancer cells on the skin for up to 18 hours. Actinic keratoses, which are precancerous growths that can evolve into squamous cell carcinomas, are the most common target for this treatment.

Conflict of Interest



  1. Hussain, Syed T., Nabin K. Shrestha, James Witten, and Steven M. Gordon, et al. "Rarity of invasiveness in right-sided infective endocarditis." J Thorac Cardiovasc Surg 155 (2018): 54-61.
  2. Google Scholar, Crossref, Indexed at

  3. López, Javier, Ana Revilla, Isidre Vilacosta, and Eduardo Villacorta, et al. "Definition, clinical profile, microbiological spectrum, and prognostic factors of early-onset prosthetic valve endocarditis." Eur Heart J 28 (2007): 760-765.
  4. Google Scholar, Crossref, Indexed at

  5. Rutledge, Robert, B. Justin Kim, and Robert E. Applebaum. "Actuarial analysis of the risk of prosthetic valve endocarditis in 1,598 patients with mechanical and bioprosthetic valves." Arch Surg 120 (1985): 469-472.
  6. Google Scholar, Crossref, Indexed at

  7. Bloomfield, Peter, David J. Wheatley, Robin J. Prescott, and Hugh C. Miller. "Twelve-year comparison of a Bjork–Shiley mechanical heart valve with porcine bioprostheses." N Engl J Med 324 (1991): 573-579.
  8. Google Scholar, Crossref, Indexed at

  9. Østergaard, Lauge, Nana Valeur, Nikolaj Ihlemann, and Henning Bundgaard, et al. "Incidence of infective endocarditis among patients considered at high risk." Eur Heart J 39 (2018): 623-629.
  10. Google Scholar, Crossref, Indexed at

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