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Covid-19 Overview
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Journal of Lung Diseases & Treatment

ISSN: 2472-1018

Open Access

Commentary - (2022) Volume 8, Issue 1

Covid-19 Overview

Priya G*
*Correspondence: Priya G, Department of Pulmonary Medicine, Osmania University, Hyderabad, Telangana, India, Email:
Department of Pulmonary Medicine, Osmania University, Hyderabad, Telangana, India

Received: 03-Jan-2022, Manuscript No. LDT-22-55705; Editor assigned: 05-Jan-2022, Pre QC No. P-55705; Reviewed: 19-Jan-2022, QC No. Q-55705; Revised: 25-Jan-2022, Manuscript No. R-55705; Published: 02-Feb-2022 , DOI: 10.37421/2472-1018.22.8.140
Citation: G, Priya. “Covid-19 Overview” J Lung Dis Treat 8 (2022): 140.
Copyright: © 2022 Priya G. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Commentary

While the Covid-19 pandemic sweeps the globe, it's critical to understand the virus's transmission and effects, which have made it a pandemic. The coronavirus family contains the SARS (Severe Acute Respiratory Syndrome) and MERS (Middle East Respiratory Symptoms) viruses, among others. The viral strains that cause the common cold and flu belong to the Coronavirus family. Covid-19 has been discovered to be a close cousin of SARS. Covid-19, like most SARS viruses, attacks the respiratory system in humans. SARS is a unique form of virus that was first described in 2007. The infection begins with flu-like symptoms or no symptoms at all, and then progresses to severe symptoms [1].

Covid-19 mainly attacks the lungs of those infected, resulting in mortality from ARDS and pneumonia in extreme instances. It's crucial to realize that it doesn't always result in ARDS and pneumonia, which is a possibility in the most severe instances [2]. In the vast majority of instances, 80 percent will have minor symptoms, 14 percent will have pneumonia, 5% will have septic shock and organ failure (mainly respiratory failure), and 2% of cases will be deadly. Fever, dizziness, dyspnea, headache, dry cough (which can lead to phlegm) and, in some cases, loss of smell and taste are some of the key symptoms to watch for in a Covid-19 infected individual. Diarrhea and weariness have also been noted in a few cases.

While a person infected with Covid-19 can be treated, it's crucial to remember that underlying conditions (hypertension, diabetes, heart difficulties, and pulmonary disorders) and those using immune-suppressing medicines are among the reasons that might lead to death. The danger is increased in older people since their immunity declines with age and they are more susceptible to various ailments. Another crucial component of comprehending Covid-19 is its impact on the human body, particularly the lungs [3,4].

It's crucial to understand the different stages/categories of Covid-19 infection before determining the severity of the illness. First, these people are infected with the virus and function as carriers, but they may not show any symptoms. These people are more likely to spread the virus since they may be unaware of its presence. Individuals with a minor fever, cough, headache, or probable conjunctivitis fall into the second group. An infection in the upper respiratory tract is to blame [5].

References

  1. Todd, Jamie L., Rahil Jain, Elizabeth N. Pavlisko and C. Ashley Finlen Copeland, et al. "Impact of forced vital capacity loss on survival after the onset of chronic lung allograft dysfunction." Am J Respir Crit Care Med 189 (2014): 159-166.
  2. Google Scholar, Crossref, Indexed at

  3. Verleden, Geert M., Ganesh Raghu, Keith C. Meyer and Allan R. Glanville, et al. "A new classification system for chronic lung allograft dysfunction." J Heart Lung Transplant 33 (2014): 127-133.
  4. Google Scholar, Crossref, Indexed at

  5. Oishi, Hisashi, Tereza Martinu, Masaaki Sato and Yasushi Matsuda, et al. "Halofuginone treatment reduces interleukin-17A and ameliorates features of chronic lung allograft dysfunction in a mouse orthotopic lung transplant model." J Heart Lung Transplant 35 (2016): 518- 527.
  6. Google Scholar, Crossref, Indexed at

  7. Wu, Qiang, Pawan Kumar Gupta, Hidemi Suzuki and Sarah R. Wagner, et al. "CD4 T cells but not Th17 cells are required for mouse lung transplant obliterative bronchiolitis." Am J Transplant 15 (2015): 1793-1804.
  8. Google Scholar, Crossref, Indexed at

  9. Gupta, Pawan K., Sarah R. Wagner, Qiang Wu, and Rebecca A. Shilling. "Th17 cells are not required for maintenance of IL‐17A‐producing γδ T cells in vivo." Immunol Cell Biol 95 (2016): 280-286.
  10. Google Scholar, Crossref, Indexed at

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