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Viral Gene Therapy Impact Factor | Open Access Journals
Molecular and Genetic Medicine

Molecular and Genetic Medicine

ISSN: 1747-0862

Open Access
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Viral Gene Therapy Impact Factor

The journal impact factor provides a quantitative assessment tool for classifying, evaluating, sorting and comparing similar journals. It reflects the average number of citations of recent articles published in scientific and social sciences journals in a given year or period and is often used as a proxy for the relative importance of a journal in its field. It was first conceived by Eugene Garfield, the founder of the Institute for Scientific Information. The impact factor of a magazine is assessed by dividing the number of citations of the current year by the elements of origin published in the magazine in the previous two years. The potential utility of viral gene replacement therapy in human diseases has been an exciting and widely studied topic in the field of skeletal muscle disease for the past 20 years. Unfortunately, while the mutant gene replacement or integration strategy is a simple concept, a number of complicating factors have been identified as gene therapy studies have progressed towards clinical trials. While some of these problems, including immunological response to viral vectors, are problems encountered with all forms of gene therapy, the use of gene therapy in skeletal muscle also poses additional challenges for which treatment strategies need to be optimized. . Despite these challenges, in recent years there has been substantial progress towards optimizing viral gene therapy for skeletal muscle disease in animal models, with an eye to optimizing the safety and efficacy of viral gene therapy in humans. Skeletal muscle disorders represent significant opportunities for investigators who develop viral gene therapy strategies, due to the numerous monogenetic diseases of skeletal muscle, clear and quantifiable clinical phenotypes and the easy accessibility of muscle tissue. Mutations in the dystrophin gene predispose the myofibre membrane to membrane damage induced by contraction, resulting in inflammation, myonecrosis and progressive loss of functional muscle tissue. Different mouse and canine models of dystrophinopathy have been used to study these diseases and evaluate potential therapies, including different viral gene therapy approaches.

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Citations: 3919

Molecular and Genetic Medicine received 3919 citations as per Google Scholar report

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