Conversations about the cause and treatment of essential hypertension usually focus on mechanisms such as sodium / volume and the renin-angiotensin system. Hypertension caused by the sympathetic nervous system, i.e. neurogenic hypertension, is less often discussed. In this audit, discuss the pathophysiology of neurogenic hypertension, the controversy between renal and central origin, the clinical records that propose neurogenic hypertension and the interventions best suited to its treatment. Neurogenic hypertension is more likely to occur in patients with labile or paroxysmal hypertension, but signs of increased sympathetic tone also suggest a neurogenic component of hypertension in patients with extreme or resistant hypertension, chronic kidney disease, comorbidities associated with increased sympathetic tone, and ingestion of medications that stimulate sympathetic tone. The significance of combined alpha and beta blockade in pharmacological treatment and the state of renal denervation are discussed. Although there are many things that are not clear in its pathophysiology, the recognition of neurogenic hypertension is of considerable clinical importance in the individualization of drug therapy and the control of blood pressure.
Case Report: Neurological Disorders
Case Report: Neurological Disorders
Case Report: Neurological Disorders
Case Report: Neurological Disorders
Editorial: Neurological Disorders
Editorial: Neurological Disorders
Letter to Editor: Neurological Disorders
Letter to Editor: Neurological Disorders
Review Article: Neurological Disorders
Review Article: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Posters & Accepted Abstracts: Neurological Disorders
Keynote: Neurological Disorders
Keynote: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
Scientific Tracks Abstracts: Neurological Disorders
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