Epithelial-mesenchymal change (EMT) is a physiological procedure wherein epithelial cells secure the motile and obtrusive attributes of mesenchymal cells. In spite of the fact that EMT in undeveloped advancement is a planned, sorted out procedure including association between a wide range of cells and tissue types, parts of the EMT program can be improperly enacted in light of micro environmental modifications and variant boosts, and this can add to ailment conditions including tissue fibrosis and malignant growth movement. Here we will diagram how EMT works in ordinary turn of events, how it could be actuated in pathologic conditions—particularly by framework metalloproteinases—and how it might be focused for remedial advantage.
Letter to Editor: Human Genetics & Embryology
Letter to Editor: Human Genetics & Embryology
Commentary: Human Genetics & Embryology
Commentary: Human Genetics & Embryology
Review Article: Human Genetics & Embryology
Review Article: Human Genetics & Embryology
Rapid Communication: Human Genetics & Embryology
Rapid Communication: Human Genetics & Embryology
Research Article: Human Genetics & Embryology
Research Article: Human Genetics & Embryology
Posters & Accepted Abstracts: Molecular and Genetic Medicine
Posters & Accepted Abstracts: Molecular and Genetic Medicine
Keynote: Molecular and Genetic Medicine
Keynote: Molecular and Genetic Medicine
Posters-Accepted Abstracts: Molecular and Genetic Medicine
Posters-Accepted Abstracts: Molecular and Genetic Medicine
Keynote: Molecular Biomarkers & Diagnosis
Posters & Accepted Abstracts: Journal of Tissue Science and Engineering
Posters & Accepted Abstracts: Journal of Tissue Science and Engineering
Human Genetics & Embryology received 309 citations as per Google Scholar report
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