Obstructive Sleep Apnea (OSA) is a pervasive sleep disorder affecting millions worldwide, characterized by disrupted breathing patterns during sleep that lead to a spectrum of health complications. A particularly intriguing facet of OSA revolves around its possible influence on exercise capacity, which denotes the aptitude to engage in physical activities. Although researchers have noted a decline in exercise capacity among individuals with OSA, the underlying mechanism driving this phenomenon has remained elusive. This article immerses itself in the intricate interplay linking OSA and the attenuation of exercise performance, venturing into a fresh hypothesis that casts light on potential contributors like endothelial dysfunction and exercise-induced pulmonary hypertension. Individuals grappling with OSA frequently encounter daytime weariness, diminished attentiveness and an overwhelming sensation of tiredness.
HTML PDFShare this article
Journal of Hypertension: Open Access received 614 citations as per Google Scholar report
